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- j.prostaglandins.2011.08.004 author "Greene ER, Huang S, Serhan CN, Panigrahy D".
- j.prostaglandins.2011.08.004 date "November 2011".
- j.prostaglandins.2011.08.004 doi "10.1016/j.prostaglandins.2011.08.004".
- j.prostaglandins.2011.08.004 isCitedBy Eoxin.
- j.prostaglandins.2011.08.004 isCitedBy Prostate_cancer.
- j.prostaglandins.2011.08.004 issue "1-4".
- j.prostaglandins.2011.08.004 issue "1–4".
- j.prostaglandins.2011.08.004 journal "Prostaglandins Other Lipid Mediat.".
- j.prostaglandins.2011.08.004 pages "27–36".
- j.prostaglandins.2011.08.004 pmc "4051344".
- j.prostaglandins.2011.08.004 pmid "21864702".
- j.prostaglandins.2011.08.004 quote "450.0".
- j.prostaglandins.2011.08.004 quote "The 5-lipoxygenase pathway is implicated in the development and progression of human cancers. 5-LOX, whose crystal structure was recently identified , is a key enzyme in metabolizing arachidonic acid to leukotrienes. 5-LOX can be induced by pro-inflammatory stimuli and is expressed in epithelial cancers including lung, prostate, breast, and colon . Hence, 5-LOX inhibitors have been targeted for their chemopreventive effects. Inhibition of 5-LOX activity is shown to block prostate cancer cell proliferation as well as carcinogen-induced lung tumorigenesis . ... Both 5-HETE and 12-HETE are also products of lipoxygenase and are involved in tumor progression . Exogenous 5-HETE can stimulate the proliferation of prostate cancer cells and act as a survival factor . These results require relatively high concentrations . Blocking the formation of 5-HETE, by inhibiting 5-lipoxygenase, results in massive apoptosis of human prostate cancer cells .".
- j.prostaglandins.2011.08.004 title "Regulation of inflammation in cancer by eicosanoids".
- j.prostaglandins.2011.08.004 volume "96".