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- j.neubiorev.2013.06.011 author "Lynch WJ, Peterson AB, Sanchez V, Abel J, Smith MA".
- j.neubiorev.2013.06.011 date "September 2013".
- j.neubiorev.2013.06.011 doi "10.1016/j.neubiorev.2013.06.011".
- j.neubiorev.2013.06.011 isCitedBy Addiction.
- j.neubiorev.2013.06.011 isCitedBy Amphetamine.
- j.neubiorev.2013.06.011 isCitedBy Neurobiological_effects_of_physical_exercise.
- j.neubiorev.2013.06.011 issue "8".
- j.neubiorev.2013.06.011 journal "Neurosci Biobehav Rev".
- j.neubiorev.2013.06.011 journal "Neurosci. Biobehav. Rev.".
- j.neubiorev.2013.06.011 pages "1622–1644".
- j.neubiorev.2013.06.011 pages "1622–44".
- j.neubiorev.2013.06.011 pmc "3788047".
- j.neubiorev.2013.06.011 pmid "23806439".
- j.neubiorev.2013.06.011 quote "These findings suggest that exercise may “magnitude”-dependently prevent the development of an addicted phenotype possibly by blocking/reversing behavioral and neuroadaptive changes that develop during and following extended access to the drug. ... Exercise has been proposed as a treatment for drug addiction that may reduce drug craving and risk of relapse. Although few clinical studies have investigated the efficacy of exercise for preventing relapse, the few studies that have been conducted generally report a reduction in drug craving and better treatment outcomes ... Taken together, these data suggest that the potential benefits of exercise during relapse, particularly for relapse to psychostimulants, may be mediated via chromatin remodeling and possibly lead to greater treatment outcomes.".
- j.neubiorev.2013.06.011 quote "[exercise] efficacy may be related to its ability to normalize glutamatergic and dopaminergic signaling and reverse drug-induced changes in chromatin via epigenetic interactions with brain-derived neurotrophic factor in the reward pathway. ... these data show that exercise can affect dopaminergic signaling at many different levels, which may underlie its ability to modify vulnerability during drug use initiation. Exercise also produces neuroadaptations that may influence an individual's vulnerability to initiate drug use. Consistent with this idea, chronic moderate levels of forced treadmill running blocks not only subsequent methamphetamine-induced conditioned place preference, but also stimulant-induced increases in dopamine release in the and striatum . ... [These] findings indicate the efficacy of exercise at reducing drug intake in drug-dependent individuals ... wheel running [reduces] methamphetamine self-administration under extended access conditions ... These findings suggest that exercise may "magnitude"-dependently prevent the development of an addicted phenotype possibly by blocking/reversing behavioral and neuro-adaptive changes that develop during and following extended access to the drug. ... Exercise has been proposed as a treatment for drug addiction that may reduce drug craving and risk of relapse. Although few clinical studies have investigated the efficacy of exercise for preventing relapse, the few studies that have been conducted generally report a reduction in drug craving and better treatment outcomes . ... Taken together, these data suggest that the potential benefits of exercise during relapse, particularly for relapse to psychostimulants, may be mediated via chromatin remodeling and possibly lead to greater treatment outcomes.".
- j.neubiorev.2013.06.011 quote "exercise can affect dopaminergic signaling at many different levels, which may underlie its ability to modify vulnerability during drug use initiation. Exercise also produces neuroadaptations that may influence an individual's vulnerability to initiate drug use. ... Exercise has been proposed as a treatment for drug addiction that may reduce drug craving and risk of relapse. Although few clinical studies have investigated the efficacy of exercise for preventing relapse, the few studies that have been conducted generally report a reduction in drug craving and better treatment outcomes ... Taken together, these data suggest that the potential benefits of exercise during relapse, particularly for relapse to psychostimulants, may be mediated via chromatin remodeling and possibly lead to greater treatment outcomes.".
- j.neubiorev.2013.06.011 title "Exercise as a novel treatment for drug addiction: a neurobiological and stage-dependent hypothesis".
- j.neubiorev.2013.06.011 volume "37".