Matches in DBpedia 2016-04 for { <http://doi.org/10.2174/157488707779318107> ?p ?o }
Showing triples 1 to 23 of
23
with 100 triples per page.
- 157488707779318107 author "Berry MD".
- 157488707779318107 date "January 2007".
- 157488707779318107 doi "10.2174/157488707779318107".
- 157488707779318107 first "MD".
- 157488707779318107 isCitedBy Attention_deficit_hyperactivity_disorder.
- 157488707779318107 isCitedBy Euphoria.
- 157488707779318107 isCitedBy Neurobiological_effects_of_physical_exercise.
- 157488707779318107 isCitedBy Phenethylamine.
- 157488707779318107 isCitedBy Physical_exercise.
- 157488707779318107 isCitedBy Trace_amine.
- 157488707779318107 issue "1".
- 157488707779318107 journal "Rev Recent Clin Trials".
- 157488707779318107 journal "Reviews on recent clinical trials".
- 157488707779318107 last "Berry".
- 157488707779318107 pages "3–19".
- 157488707779318107 pmid "18473983".
- 157488707779318107 quote "It has also been suggested that the antidepressant effects of exercise are due to an exercise-induced elevation of PE [151].".
- 157488707779318107 quote "More direct evidence has been obtained recently for a role of trace amines in ADHD. Urinary PE levels have been reported to be decreased in ADHD patients in comparison to both controls and patients with autism [103-105]. Evidence for a decrease in PE levels in the brain of ADHD patients has also recently been reported [4]. In addition, decreases in the urine and plasma levels of the PE metabolite phenylacetic acid and the precursors phenylalanine and tyrosine have been reported along with decreases in plasma tyramine [103]. Following treatment with methylphenidate, patients who responded positively showed a normalization of urinary PE, whilst non-responders showed no change from baseline values [105].".
- 157488707779318107 quote "changes in trace amines, in particular PE, have been identified as a possible factor for the onset of attention deficit/hyperactivity disorder [5, 27, 43, 78]. PE has been shown to induce hyperactivity and aggression, two of the cardinal clinical features of ADHD, in experimental animals [100]. Hyperactivity is also a symptom of phenylketonuria, which as discussed above is associated with a markedly elevated PE turnover [44]. Further, amphetamines, which have clinical utility in ADHD, are good ligands at trace amine receptors [2]. Of possible relevance in this aspect is modafanil, which has shown beneficial effects in ADHD patients [101] and has been reported to enhance the activity of PE at TAAR1 [102]. Conversely, methylphenidate, which is also clinically useful in ADHD, showed poor efficacy at the TAAR1 receptor [2]. In this respect it is worth noting that the enhancement of functioning at TAAR1 seen with modafanil was not a result of a direct interaction with TAAR1 [102].".
- 157488707779318107 title "The potential of trace amines and their receptors for treating neurological and psychiatric diseases".
- 157488707779318107 vauthors "Berry MD".
- 157488707779318107 volume "2".
- 157488707779318107 year "2007".