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- j.tips.2005.03.007 author "Lindemann L, Hoener MC".
- j.tips.2005.03.007 author "Lindemann, L.; Hoener, M. C.".
- j.tips.2005.03.007 date "May 2005".
- j.tips.2005.03.007 doi "10.1016/j.tips.2005.03.007".
- j.tips.2005.03.007 first1 "Lothar".
- j.tips.2005.03.007 first2 "Marius C.".
- j.tips.2005.03.007 isCitedBy Amphetamine.
- j.tips.2005.03.007 isCitedBy Euphoria.
- j.tips.2005.03.007 isCitedBy N-Methylphenethylamine.
- j.tips.2005.03.007 isCitedBy N-Methyltyramine.
- j.tips.2005.03.007 isCitedBy Neurobiological_effects_of_physical_exercise.
- j.tips.2005.03.007 isCitedBy Phenethylamine.
- j.tips.2005.03.007 isCitedBy Physical_exercise.
- j.tips.2005.03.007 isCitedBy Synephrine.
- j.tips.2005.03.007 isCitedBy Trace_amine.
- j.tips.2005.03.007 isCitedBy Trace_amine-associated_receptor.
- j.tips.2005.03.007 issue "5".
- j.tips.2005.03.007 journal "Trends Pharmacol. Sci.".
- j.tips.2005.03.007 journal "Trends in Pharmacological Sciences".
- j.tips.2005.03.007 last1 "Lindemann".
- j.tips.2005.03.007 last2 "Hoener".
- j.tips.2005.03.007 pages "274–281".
- j.tips.2005.03.007 pages "274–81".
- j.tips.2005.03.007 pmid "15860375".
- j.tips.2005.03.007 quote "30.0".
- j.tips.2005.03.007 quote "In addition to the main metabolic pathway, TAs can also be converted by nonspecific N-methyltransferase [22] and phenylethanolamine N-methyltransferase [23] to the corresponding secondary amines , which display similar activities on TAAR1 as their primary amine precursors.".
- j.tips.2005.03.007 quote "In addition to the main metabolic pathway, TAs can also be converted by nonspecific N-methyltransferase [22] and phenylethanolamine N-methyltransferase [23] to the corresponding secondary amines , which display similar activities on TAAR1 as their primary amine precursors...Both dopamine and 3-methoxytyramine, which do not undergo further N-methylation, are partial agonists of TAAR1 . ...".
- j.tips.2005.03.007 quote "The dysregulation of TA levels has been linked to several diseases, which highlights the corresponding members of the TAAR family as potential targets for drug development. In this article, we focus on the relevance of TAs and their receptors to nervous system-related disorders, namely schizophrenia and depression; however, TAs have also been linked to other diseases such as migraine, attention deficit hyperactivity disorder, substance abuse and eating disorders [7,8,36]. Clinical studies report increased β-PEA plasma levels in patients suffering from acute schizophrenia [37] and elevated urinary excretion of β-PEA in paranoid schizophrenics [38], which supports a role of TAs in schizophrenia. As a result of these studies, β-PEA has been referred to as the body’s ‘endogenous amphetamine’ [39]".
- j.tips.2005.03.007 title "A renaissance in trace amines inspired by a novel GPCR family".
- j.tips.2005.03.007 url S0165-6147(05)00079-9.
- j.tips.2005.03.007 vauthors "Lindemann L, Hoener MC".
- j.tips.2005.03.007 volume "26".
- j.tips.2005.03.007 year "2005".