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- j.pharmthera.2009.11.005 author "Broadley KJ".
- j.pharmthera.2009.11.005 date "Mar 2010".
- j.pharmthera.2009.11.005 date "March 2010".
- j.pharmthera.2009.11.005 doi "10.1016/j.pharmthera.2009.11.005".
- j.pharmthera.2009.11.005 first "KJ".
- j.pharmthera.2009.11.005 isCitedBy Amphetamine.
- j.pharmthera.2009.11.005 isCitedBy N-Methylphenethylamine.
- j.pharmthera.2009.11.005 isCitedBy N-Methyltyramine.
- j.pharmthera.2009.11.005 isCitedBy Neurobiological_effects_of_physical_exercise.
- j.pharmthera.2009.11.005 isCitedBy Nutritional_neuroscience.
- j.pharmthera.2009.11.005 isCitedBy Olfactory_receptor.
- j.pharmthera.2009.11.005 isCitedBy Phenethylamine.
- j.pharmthera.2009.11.005 isCitedBy Stimulant.
- j.pharmthera.2009.11.005 isCitedBy Trace_amine.
- j.pharmthera.2009.11.005 issue "3".
- j.pharmthera.2009.11.005 journal "Pharmacol. Ther.".
- j.pharmthera.2009.11.005 journal "Pharmacology & Therapeutics".
- j.pharmthera.2009.11.005 journal "Pharmacology & therapeutics".
- j.pharmthera.2009.11.005 last "Broadley".
- j.pharmthera.2009.11.005 pages "363–375".
- j.pharmthera.2009.11.005 pages "363–75".
- j.pharmthera.2009.11.005 pmid "19948186".
- j.pharmthera.2009.11.005 quote "Brain levels of endogenous trace amines are several hundred-fold below those for the classical neurotransmitters noradrenaline, dopamine and serotonin but their rates of synthesis are equivalent to those of noradrenaline and dopamine and they have a very rapid turnover rate . Endogenous extracellular tissue levels of trace amines measured in the brain are in the low nanomolar range. These low concentrations arise because of their very short half-life ...".
- j.pharmthera.2009.11.005 quote "Brain levels of endogenous trace amines are several hundred-fold below those for the classical neurotransmitters noradrenaline, dopamine and serotonin but their rates of synthesis are equivalent to those of noradrenaline and dopamine and they have a very rapid turnover rate . Endogenous extracellular tissue levels of trace amines measured in the brain are in the low nanomolar range. These low concentrations arise because of their very short half-life,...".
- j.pharmthera.2009.11.005 quote "Fig. 2. Synthetic and metabolic pathways for endogenous and exogenously administered trace amines and sympathomimetic amines ...".
- j.pharmthera.2009.11.005 quote "Fig. 2. Synthetic and metabolic pathways for endogenous and exogenously administered trace amines and sympathomimetic amines...".
- j.pharmthera.2009.11.005 quote "Thus, MAO inhibitors potentiate the peripheral effects of indirectly acting sympathomimetic amines ... this potentiation occurs irrespective of whether the amine is a substrate for MAO. An α-methyl group on the side chain, as in amphetamine and ephedrine, renders the amine immune to deamination so that they are not metabolized in the gut. Similarly, β-PEA would not be deaminated in the gut as it is a selective substrate for MAO-B which is not found in the gut ...".
- j.pharmthera.2009.11.005 quote "Thus, MAO inhibitors potentiate the peripheral effects of indirectly acting sympathomimetic amines. It is not often realized, however, that this potentiation occurs irrespective of whether the amine is a substrate for MAO. An α-methyl group on the side chain, as in amphetamine and ephedrine, renders the amine immune to deamination so that they are not metabolized in the gut. Similarly, β-PEA would not be deaminated in the gut as it is a selective substrate for MAO-B which is not found in the gut...".
- j.pharmthera.2009.11.005 quote "Trace amines are metabolized in the mammalian body via monoamine oxidase ... It deaminates primary and secondary amines that are free in the neuronal cytoplasm but not those bound in storage vesicles of the sympathetic neurone ... Similarly, β-PEA would not be deaminated in the gut as it is a selective substrate for MAO-B which is not found in the gut ...".
- j.pharmthera.2009.11.005 quote "Trace amines are metabolized in the mammalian body via monoamine oxidase ... It deaminates primary and secondary amines that are free in the neuronal cytoplasm but not those bound in storage vesicles of the sympathetic neurone ...".
- j.pharmthera.2009.11.005 quote "Trace amines are metabolized in the mammalian body via monoamine oxidase ...It deaminates primary and secondary amines that are free in the neuronal cytoplasm but not those bound in storage vesicles of the sympathetic neurone...".
- j.pharmthera.2009.11.005 title "The vascular effects of trace amines and amphetamines".
- j.pharmthera.2009.11.005 title "The vascular effects of trace amines and amphetamines.".
- j.pharmthera.2009.11.005 vauthors "Broadley KJ".
- j.pharmthera.2009.11.005 volume "125".