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- j.brainresrev.2009.03.002 author "Krasnova IN, Cadet JL".
- j.brainresrev.2009.03.002 date "May 2009".
- j.brainresrev.2009.03.002 doi "10.1016/j.brainresrev.2009.03.002".
- j.brainresrev.2009.03.002 isCitedBy Amphetamine_dependence.
- j.brainresrev.2009.03.002 isCitedBy Methamphetamine.
- j.brainresrev.2009.03.002 isCitedBy Stimulant.
- j.brainresrev.2009.03.002 issue "2".
- j.brainresrev.2009.03.002 journal "Brain Res Rev".
- j.brainresrev.2009.03.002 journal "Brain Res. Rev.".
- j.brainresrev.2009.03.002 pages "379–407".
- j.brainresrev.2009.03.002 pages "379–407".
- j.brainresrev.2009.03.002 pmc "2731235".
- j.brainresrev.2009.03.002 pmid "19328213".
- j.brainresrev.2009.03.002 quote "Neuroimaging studies have revealed that METH can indeed cause neurodegenerative changes in the brains of human addicts . These abnormalities include persistent decreases in the levels of dopamine transporters in the orbitofrontal cortex, dorsolateral prefrontal cortex, and the caudate-putamen . The density of serotonin transporters is also decreased in the midbrain, caudate, putamen, hypothalamus, thalamus, the orbitofrontal, temporal, and cingulate cortices of METH-dependent individuals ...".
- j.brainresrev.2009.03.002 quote "Neuroimaging studies have revealed that METH can indeed cause neurodegenerative changes in the brains of human addicts . These abnormalities include persistent decreases in the levels of dopamine transporters in the orbitofrontal cortex, dorsolateral prefrontal cortex, and the caudate-putamen . The density of serotonin transporters is also decreased in the midbrain, caudate, putamen, hypothalamus, thalamus, the orbitofrontal, temporal, and cingulate cortices of METH-dependent individuals .".
- j.brainresrev.2009.03.002 quote "Neuropsychological studies have detected deficits in attention, working memory, and decision-making in chronic METH addicts ...".
- j.brainresrev.2009.03.002 quote "Structural magnetic resonance imaging studies in METH addicts have revealed substantial morphological changes in their brains. These include loss of gray matter in the cingulate, limbic and paralimbic cortices, significant shrinkage of hippocampi, and hypertrophy of white matter . In addition, the brains of METH abusers show evidence of hyperintensities in white matter , decreases in the neuronal marker, N-acetylaspartate , reductions in a marker of metabolic integrity, creatine and increases in a marker of glial activation, myoinositol . Elevated choline levels, which are indicative of increased cellular membrane synthesis and turnover are also evident in the frontal gray matter of METH abusers .".
- j.brainresrev.2009.03.002 quote "Structural magnetic resonance imaging studies in METH addicts have revealed substantial morphological changes in their brains. These include loss of gray matter in the cingulate, limbic, and paralimbic cortices, significant shrinkage of hippocampi, and hypertrophy of white matter . In addition, the brains of METH abusers show evidence of hyperintensities in white matter , decreases in the neuronal marker, N-acetylaspartate , reductions in a marker of metabolic integrity, creatine and increases in a marker of glial activation, myoinositol . Elevated choline levels, which are indicative of increased cellular membrane synthesis and turnover are also evident in the frontal gray matter of METH abusers .".
- j.brainresrev.2009.03.002 quote "There is compelling evidence that the negative neuropsychiatric consequences of METH abuse are due, at least in part, to drug-induced neuropathological changes in the brains of these METH-exposed individuals ...".
- j.brainresrev.2009.03.002 title "Methamphetamine toxicity and messengers of death".
- j.brainresrev.2009.03.002 volume "60".